Molecular Vision: Li, Mol Vis 2008; 14:750-755. Table 1

Table 1. Clinical features of affected family members.

Number	Age	Gender	Onset age	Surgery age	Clinical features
II1	73	Female	20	30	Aphakia, after cataract surgery
II3	61	Male	14–15	30	Aphakia, after cataract surgery
III1	53	Male	30		Bilateral coronary,punctate, asteroidal (above the posterior pole) cataracts
III2	50	Male	20		Bilateral coronary, punctate cataracts; gray opacity in partial coronary area
III3	42	Female	15	25	Aphakia, after cataract surgery
III4	47	Female	16–17	32	Right coronary, punctate, asteroidal (posterior pole) cataracts; left after cataract surgery
III5	36	Female	14–15	27	Aphakia, after cataract surgery
III20	40	Female	13	22	After bilateral phacoemulsification and intraocular lens implantation
III21	37	Female	10	29	After bialateral phacoemulsification and intraocular lens implantation
III22	35	Male	12		Bilateral punctate, asteroidal (above the posterior pole) cataracts
III23	33	Male	12	33	Proband
IV1	25	Male	12		Bilateral punctate, asteroidal (above the posterior pole) cataracts
IV4	22	Female	12		Bilateral punctate, asteroidal (above the posterior pole) cataracts
IV8	19	Male	10		Bilateral coronary, punctate cataracts
IV10	24	Female	15		Bilateral coronary, inverted T-shaped (posterior pole) cataracts
IV15	13	Male			Bilateral sparse punctate, asteroidal (above the posterior pole) cataracts
IV16	6	Male			Sparse punctate opacities

Before the age of 10, no clinical features were manifest (IV16); punctate opacification was primarily scattered in the lens perimeter, causing almost no influence on the life of the affected individuals. In adolescence, affected individuals showed ambiguous visual clinical features. At about the age of 30, the clinical features became serious. Two affected family members (III4, III23) had different clinical features between their two lenses; III1, III2, III22, and IV10 had different clinical features from each other. The clinical features of III22, IV1, and IV4 were similar.